Home » News » Deposit loan app that pays agents direct launches after false start previous nextProptechDeposit loan app that pays agents direct launches after false startFronted has some tech megastar co-founders and aims to increase social mobility by helping tenants pay their deposits more easily.Nigel Lewis17th February 20210474 Views A new app that aims to improve social mobility by lending rental deposits has launched after a false start last year, despite being led by some of tech’s biggest names.Fronted uses open banking and other financial technology and aims to finance deposits directly and more cheaply than existing options, such as credit cards, pay-day lenders and overdrafts, or insurance-backed membership schemes.The fintech also promises that its offer is lower risk. Once a rental loan is agreed, the money is sent directly to the estate agent to be put in the deposit protection scheme – and tenants pay back the cash in manageable amounts.Last March, co-founder and CEO Jamie Campbell (above, right) who made his name at Open Banking platform Bud decided to hibernate the new business as the pandemic took hold.With rents falling in London, it’s now the right time for the start-up to officially launch as 60% of all renters have no savings and aren’t in a great position to move, says Campbell.The other two co-founders are former Monzo high flier Simon Vans-Colina (above, centre) and Anthony Mann (left), who used to work at Apple.Social mobility“Deposits are a huge impediment to social mobility,” he explains. “People who haven’t found a place, they are coming to us to see what deposit they could get and then going to find a property.“For those people, we give them a maximum which is valid for 30 days so they can shop around knowing they have Fronted in their back pocket.”To apply for a rental deposit loan, tenants need to be a UK citizen, have a bank account with more than six months of transaction data and a minimum income of £12,000.The app then checks affordability beyond a simple credit score; those on benefits or furloughed will also be assessed. Loans last 12 months, carry a 12.5% interest rate and no early repayment fees.Fronted Jamie Campbell tenant deposits February 17, 2021Nigel LewisWhat’s your opinion? Cancel replyYou must be logged in to post a comment.Please note: This is a site for professional discussion. Comments will carry your full name and company.This site uses Akismet to reduce spam. Learn how your comment data is processed.Related articles BREAKING: Evictions paperwork must now include ‘breathing space’ scheme details30th April 2021 City dwellers most satisfied with where they live30th April 2021 Hong Kong remains most expensive city to rent with London in 4th place30th April 2021
Read more posts about Governor Wolf’s 2016-17 budget.Like Governor Tom Wolf on Facebook: Facebook.com/GovernorWolf Budget News, Government That Works, Press Release Governor Wolf remains committed to improving efficiencies, streamlining government operations, and reducing costs. He continues to change the way business is conducted to ensure that taxpayer dollars are being spent wisely, redundancies are eliminated, and customer interactions at all levels are improved.Just last week, Governor Wolf gave an update on these improvements, announcing produced savings for both the current and future state budgets. The Wolf Administration is on track to save $150 million in the current fiscal year. To date, GO-TIME has identified nearly 250 initiatives by state agencies to modernize government operations by increasing efficiency, improving technology, and identifying opportunities for agencies to collaborate and share resources. These initiatives will provide benefits including savings, reduced processing times, increased convenience, better customer service, and maximizing existing resources.Last year, Governor Wolf’s administration implemented many reforms that generated substantial taxpayer savings and increased service effectiveness, including: eliminating the SNAP asset test, reducing the food stamp error rate, helping more TANF families find employment, lowering insurance rates by more than $80 million, and reducing the prison population the largest amount in 40 years.The 2016-17 Budget will again focus on creating a government that works effectively through innovation and increased efficiencies to ensure that taxpayer dollars are wisely spent.Reducing Redundancy and Improving OperationsThis budget includes the merger of the Department of Corrections and the Board of Probation and Parole and the relocation of the eHealth Partnership Authority to within the Department of Human Services. These steps continue the work Governor Wolf has taken throughout the course of the last year to ensure that the commonwealth truly is a government that works.Ensuring Taxpayers are Only Paying for SuccessThe 2016-2017 budget increases the use of Pay for Success performance contracts, which provide an innovative strategy to finance proven programs through public-private partnerships on a straightforward proposition. The Wolf Administration will work with private sector partners to address the employment needs of individuals with criminal convictions to make them less likely to return to the corrections system and more likely to build a foundation for a stable, productive life. Additionally, the administration will engage youth advocate partners to provide intensive support to juvenile offenders and their families in their homes, schools, and communities to provide resources, skill development and educational and vocational work.Taxpayers will only pay for services that get results and save money in the long run. Payments are tied to the achievement of specific, pre-established performance goals that are set forth in the contract, and only occur after rigorous evaluation.Making Pensions Improvements Beyond Act 120The 2016-17 year will mark the end of steep increases in state and local school district contributions to pay unfunded future pension obligations. State General Fund costs will increase by nearly $500 million before beginning to level off in future years. While the General Assembly debates further changes to benefit plans, several administrative and financial management initiatives can reduce future obligations, includingConsolidating investment management operations among the funds that manage short- and long-term investment assetsReducing fees for investment management servicesEstablishing a restricted receipt account to ensure full funding of future pension obligations February 09, 2016 Budget Briefing: Government that Works – Innovation and Efficiency SHARE Email Facebook Twitter
But the addition of Kaymer and Donald means the tournament will feature five players who have been world number one – McIlroy, Donald, Kaymer, Ernie Els and Lee Westwood – and eight major champions in McIlroy, Els, Kaymer, Darren Clarke, Padraig Harrington, Graeme McDowell, Paul Lawrie and Jose Maria Olazabal. Ryder Cup captain Clarke said: “Rory has pulled in a lot of favours and it’s a testament to the high esteem Rory is held that it’s going to be the strongest field for a long time. “It’s a huge positive for the European Tour to have such a tournament on one of the best golf courses in the world. “I have always wanted to win the Irish Open but have not managed to play well in it that often. Hopefully being back at Royal County Down I will have a decent week this year.” Donald, who last played in the Irish Open in 2004, said: “My performances and results have been getting better and better recently and I am feeling very positive about a huge summer of golf ahead. “The Irish Open will be a big part of that and I am looking forward to playing Royal County Down and some really ‘old school’ links golf – I think the links golf factor will add a lot to the event and it’s looking like it’s going to be a great week.” Kaymer added: “There is definitely a good feeling about this year’s Irish Open and I think Rory has done a great job to get such a great field for the event and to create a really nice atmosphere about the tournament.” The nature of the course means that spectator numbers will be limited to 20,000 a day, although 5,000 grandstand seats will be erected. Press Association Rory McIlroy’s powers of persuasion have resulted in US Open champion Martin Kaymer, former world number one Luke Donald and American Ryder Cup star Patrick Reed in confirming their participation in the Dubai Duty Free Irish Open. McIlroy’s foundation is hosting the event at Royal County Down from May 28-31 and the world number one has been a major influence in attracting a top-class field, who will compete for a prize fund of 2.5million euro (£1.8million), an increase of 25 per cent from last year. Reed let the cat out of the bag on the final day of the Masters at Augusta National, also revealing he would compete in the BMW PGA Championship at Wentworth the week before.
MLB playoffs 2019: One reason Nationals, Brewers could win the World Series Related News THEY DON’T CALL IT THE MILD CARD, FAM. pic.twitter.com/bW4MknRhgs— Cut4 (@Cut4) October 2, 2019But as amazing as his on-field excitement was, it paled in comparison to that of his father who sought him out on the field after the game to join in on the celebration.His father was so hyped up he jumped on his son’s back and dragged him onto the ground as both were laughing and smiling. MLB playoffs 2019: Brewers’ Trent Grisham doesn’t believe pressure got to him on error MLB playoffs 2019: Three takeaways from an improbable Nationals wild-card win Juan Soto was fired up Tuesday.The 20-year-old left fielder delivered a go-ahead, 3-run single in the Nationals’ 4-3 wild-card game win over the Brewers and his celebration on the field after his hit was incredible. It truly has to be seen to be fully appreciated.Juan Soto getting celebratory tackled by his dad is so amazing. pic.twitter.com/8atUzu4Ck2— Cut4 (@Cut4) October 2, 2019There’s a lot of bad going on in this world, but Soto and his father gave us a little bit of good for a few minutes.Enjoy it.
Golfers will have to sport a sweater as they tee off for the Honda Classic at PGA National in Palm Beach Gardens. High temperatures will only reach the 60’s with lows in the 30’s and 40’s…but there will be plenty of sunshine.Check out the golfers playing in the 2020 Honda Classic here.
Aliquippa (13-0) Head Coach Mike Zmijanac doesn’t keep track of career milestones. He admitted as much prior to the start of the WPIAL Championship Trophy presentation ceremony.The pleasant and unseasonably warm temperatures that gave the east coast a feeling of summer in late November might have caused some Washington (12-1) players to believe that they really had a chance to upset the Quips. ELUDES DEFENDER—Terry Swanson of Aliquippa eludes a Washington defender in the Quips 34-7 win, Swanson rushed for 206 yards. The WPIAL’s leading rusher junior Shai “Shy” McKenzie, entered the game with 2,689 yards and 42 touchdowns, but the Quips held him to only 33 yards on 18 carries. It was Aliquippa’s junior Terry Swanson who stole the show by rushing for 206 yards, including a 60 yards touchdown to lead the Quips to a 34-7 victory.The best–kept secret in football isn’t one anymore, other than perhaps if you see Swanson out of uniform.Swanson and Dravon Henry are the only set of teammates in the WPIAL to have rushed for more than 1,000 yards.Aliquippa won a record 15th WPIAL football championship. The Quips are a Class A school that plays up in classification to Class AA. The two best area teams are Aliquippa and Clairton. It would make for a dream matchup. Will they ever face one another?“We had a deal to play Clairton at the beginning of the 2012 season,” said defensive coach Greg Gill. “But, the WPIAL would not approve of the game. I hope someone can make that game happen and the Bears will find out what Aliquippa is all about.”
Ross Anderson (PhD biochemistry) is professor of biochemistry at The Master’s University in southern California. Dr Anderson’s expertise is in the area of biochemistry and molecular biology. He has taught Biochemistry and helped to direct research projects of graduate and medical students at Baylor College of Medicine, Houston, TX. Dr. Anderson was a post-doctoral researcher in the Molecular Genetics Division of the Department of Ophthalmology at the Houston Neurosensory Center.Dr Anderson was a member of both the undergraduate and graduate faculty at Lamar University, Beaumont, TX. There he taught and directed the research activities of undergraduates and Masters of Science degree candidates in Biology. Currently he is professor of biochemistry at The Master’s University in southern California.Dr Anderson’s research interests include structure-function studies of DNA polymerizing enzymes and the synthesis and expression of synthetic human genes in bacterial hosts. He has authored or co-authored several publications in major, peer-reviewed journals. He is a member of the American Chemical Society and Sigma Xi Research Society. by Ross Anderson, PhDMitochondria are very interesting and unique organelles. All eukaryotic cells possess them. They are indispensable in a number of cell activities beyond their well-known role of synthesizing the bulk of ATP used to power cells. Mitochondria reproduce at rates that are, for the most part, independent of the cell division cycle. However, they always manage to generate about twice the number just before the time of mitosis and cytokinesis so that each daughter cell receives approximately equal numbers of mitochondria. How this is regulated and coordinated is unknown.The Mitochondrial GenomeMitochondria have their own genomes separate from that in the nucleus. Each mitochondrion may have 5 to 10 copies of the genome organized into clusters called nucleoids, and a cell may have a thousand mitochondria. This means that each cell may have 5,000 to 10,000 copies of the mitochondrial genome! At conception it is estimated that mammals inherit 100,000 to 500,000 mitochondrial genomes via the egg!The Problem of Mutational DegradationDue to mutations, it is possible for a single mitochondrion to have “healthy” copies as well as defective/mutated copies. Such cells with two or more distinct varieties of mitochondrial DNA (mtDNA) are said to be heteroplasmic. Mitochondrial DNA encodes various protein subunits used for ATP production, their own transfer RNAs (tRNA), and ribosomal RNAs (rRNA). Most of the proteins used in the mitochondria, however, are encoded in nuclear genes.Like nuclear DNA, mtDNA mutates, but at a much greater rate; 5 to 10 times greater than the genes in the nucleus. This may possibly be due to lower fidelity in DNA replication, lower efficiency in DNA repair mechanisms, or both. There are a number of disease states that are the result of too many mutated mitochondrial genes.Mitochondrial InheritanceMitochondria of mammalian cells and fruit flies, indeed most eukaryotes, are inherited from the mother’s egg cells. These diseases then are inherited through the mother. Consequently, it is imperative that those mitochondria be as “healthy” genetically as possible. If a mitochondrion generates more mutated copies than can be tolerated, there must be a mechanism for recognizing these defective mitochondria and destroying them so that only “healthy” mitochondria are maintained in the egg and passed on to the next generation. The high rates of mutation would suggest that most cells would exhibit heteroplasmy, but this is not the case. Most cells are homoplastic; i.e., they have mitochondria with identical genomes. Thus, there must be a way that cells are able to identify mitochondria with defective DNA, keep them from reproducing and eliminate them. The cells, in effect, create a bottleneck through which only “healthy” mitochondria are permitted to pass.Forcing Mitochondria Through a Bottleneck InspectionThe authors of a letter in Nature by Lieber et al. performed a series of ingenious experiments investigating factors that may be involved in identifying defective mitochondria; i.e., creating the bottleneck. They transplanted wild-type mitochondria from one species of fruit fly, D. yakuba, into another species of fruit fly, D. melanogaster, which already had mitochondrial genomes with a temperature-sensitive point mutation in the cytochrome c oxidase subunit I gene. Flies grown at the permissive temperature (18○C) exhibited little effect, while those grown at the restrictive temperature (29○C) exhibited reduced cytochrome c oxidase activity.When grown at the restrictive temperature, they found there was a marked increase in the proportion of wild-type mitochondrial DNA from D. yakuba relative to the defective mtDNA, but only in the germline cells—not somatic cells. This indicated that the defective mtDNA was somehow selectively detected and destroyed.Whether from an evolutionary or a design perspective, this makes perfect sense. Somatic cells can tolerate defective mitochondria more than germline cells because only that cell would be affected; it would not necessarily impact the next generation. Germline cells, though, would carry the defect into the next generation. Not surprisingly, it was also found that detection and destruction of defective mtDNA was limited to female germline cells, not male germline cells.Selection for destruction appeared to initiate in the very early stages of egg development; after the stem cell stage and during the cyst formation stage. Further experiments revealed that selection was at the level of whole mitochondria—not just defective DNA. Thus, it was the defective mitochondria that were being detected and destroyed.How Fragmentation Rescues Healthy mtDNAThe morphology of the mitochondrion changes from the stem cell stage to the cyst stage. The authors postulate that this change is due to fragmentation of the mitochondria, such that defective mitochondria are more easily detected. Additionally, from the 2-to-8-cell stage in the cyst, mtDNA is not replicated. This reduces the number of genomes per mitochondrion and decreases the possibility of both defective and wild-type genomes residing in the same mitochondrion. This strategy leads to improved selection. Mitochondria in the stem cell stage (which occurs prior to the cyst stage) were shown to share their contents easily. Fragmentation in the cyst cell stage, however, decreases the possibility of defective and wild-type mitochondria from sharing their contents.In this respect the stem cells are more like somatic cells. The reasoning is that in somatic cells mitochondria with defective DNA can fuse and share their contents, thus a “healthy” mitochondrion can complement and rescue a mitochondrion with defective DNA. Fusion cannot be permitted in germline cells, because the defective mtDNA would be masked by the “healthy” DNA, allowing mutations to accrue and to be passed on to each subsequent generation. The authors suggest that fragmentation, in some unknown way, aids in distinguishing defective from wild-type mitochondria.Intelligent SelectionThe authors were able to show that either overexpression of the protein Mitofusin (a protein involved in the fusion of mitochondria, and suppression of fragmentation), or the reduced expression of DrpI (a protein involved in mitochondrial fission; promotes fragmentation) led to loss of selection. This, again, suggested that a time of sustained fragmentation is necessary for selection. It was shown that mitochondrial fragmentation is not only necessary, but also sufficient for effective selection against defective mtDNA.Interestingly, experimentally reduced expression of Mitofusin in somatic cells also led to sustained fragmentation and subsequent selection against defective mtDNA. However, a reduction in Mitofusin expression (both the protein and its mRNA) is normally found only in the germline cells at the cyst cell stage; i.e., this reduction in Mitofusin is observed selectively in germline cells, not somatic cells.Design in the Timing, TooATP synthase is a rotary motor that generates 3 ATP per revolution.This observation strongly suggests a design feature for the purpose of generating a period of sustained fragmentation early in egg development so that selection against defective mtDNA can be more effective. Further investigation is needed to ascertain just how defective mtDNA is identified. However, the authors were able to determine that reduction in ATP production is sufficient to induce selection. This makes sense considering that most of the protein-coding genes of mitochondria are for subunits of the ATP synthase rotary engines or for the other complex machinery of the electron transport chain.Another protein, BNIP3, was also found to be selectively upregulated in cyst cells. This protein is located in the mitochondrial outer membrane. It plays an important role in mitophagy [mitochondrial recycling] in somatic cells as when maturing red blood cells need to rid themselves of all their organelles including mitochondria.Design ThroughoutSo, the selective reduction of Mitofusin expression and increased expression of DrpI and BNIP3 at the same time and in the same select group of cells (cyst cells) smacks of design! We must also keep in mind that these proteins do not work alone, but rather in coordinated complexes with a number of other proteins designed to carry out a specific function at a specific time and place. If that’s not enough, these authors also mention reports that another protein, Pink1, may recognize and inhibit replication of the defective mtDNA, which in some unknown way stimulates replication of the wild-type DNA so that the “healthy” DNA can dominate.Design in the CleanupOnce mitochondria carrying defective DNA have been selected, they are destroyed by a process known as mitophagy, a type of autophagy whereby certain organelles are selectively destroyed, and their contents recycled by the cell. Pink1 in conjunction with another protein, Parkin, initiate mitophagy. In healthy mitochondria, Pink1 is imported into the matrix of the mitochondrion, but is not imported by defective mitochondria. Instead it accumulates on the outer surface of the outer membrane, recruits Parkin which, in turn, adds ubiquitin tags to outer membrane proteins targeting them for destruction.SummaryIt appears that the ability of a developing egg cell to create the genetic bottleneck leading to homoplastic cells is its ability to initiate sustained fragmentation whereby mitochondria containing defective DNA are selectively eliminated. Taken in toto with other information not in the article, it seems to me that the design inference is strong.This article, like virtually all others of its kind, addresses the scientific questions of ‘What is it? What does it do?’ And ‘How does it do it?’ While we slowly learn more and more answers to these questions, and someday may be able to describe in intimate detail the inner workings of cells, we will never be able to accurately answer the question of ‘How it came to be?’ short of invoking the genius of an all-powerful, creative designer.The authors, unfortunately, felt the need to attribute the process of selective identification and destruction of aberrant mtDNA to the all-powerful deity, Evolution, and thus gave their token bow. I find it simply amazing how so many intelligent researchers can report many of the detailed features of living creatures yet still attribute it to chance and state that any inference to design is only an illusion. All researchers need to take a few steps back and look at the forest, and not be so focused on the trees. (Visited 304 times, 1 visits today)FacebookTwitterPinterestSave分享0
Share Facebook Twitter Google + LinkedIn Pinterest A crush of supporters filed friend-of-the-court briefs, joining the American Farm Bureau Federation in urging the U.S. Supreme Court to hear arguments on the Environmental Protection Agency’s plan to micromanage state land-use and development decisions under the guise of the Chesapeake Bay water quality “blueprint.”Filers included 92 members of Congress, 22 states, forestry groups represented by the Pacific Legal Foundation, and a broad cross-section of the U.S. economy represented by the U.S. Chamber of Commerce, the National Association of Manufacturers and the National Federation of Independent Business.“The fact that so many voices are being raised in support of Supreme Court review shows the broad and severe threat that EPA’s action here poses nationwide,” said Bob Stallman, AFBF president. “EPA has asserted powers that do not appear in any law written by Congress, and it has done so in the context of an iconic national treasure, hoping that will inoculate its power grab in the courts. We have faith that the nation’s highest court will see this for what it is and hold EPA accountable to stay within its statutory authority.”Despite aggressive new commitments and water quality achievements by the six states in the Bay watershed in the mid-2000s, the EPA asserted federal control over the Chesapeake Bay recovery in its 2010 “blueprint.” The new federal plan effectively gives EPA the ability to function as a super-zoning authority over local and state governments—dictating where homes can be built, where land can be farmed, and where commercial development can occur.The plan will impose tens of billions of dollars in direct costs — with unknown economic impacts on local communities and economies. It also denies state and local governments and businesses the flexibility to adapt to new circumstances, instead locking in limits that can quickly become outdated but can only be revised by EPA. The lower courts upheld EPA’s blueprint on the theory that it furthers the water quality goals of the Clean Water Act — despite the absence of words in the statute authorizing such federal action. A significant issue presented for the Supreme Court is the degree to which courts should defer to broad agency interpretations of their statutory power.“The broad support for the Farm Bureau petition shows that deep concerns about the Bay blueprint go far beyond agriculture and far beyond the Bay region,” said Ellen Steen, AFBF General Counsel. “Members of Congress, states and business groups recognize that this illegal framework will be imposed throughout the country unless the Court intervenes. Given the enormous social and economic consequences, not to mention the grave questions about federalism and deference to agency overreaching, this is a case that cries out for Supreme Court review.”
About the authorCarlos VolcanoShare the loveHave your say Juventus captain Chiellini: We care about Supercoppaby Carlos Volcano9 months agoSend to a friendShare the loveJuventus captain Giorgio Chiellini says they’re determined to win the Supercoppa against Juventus tonight.Juve lost on penalties in the December 2016 Supercoppa in Doha.“We have changed a great deal in two years, but it is certainly a trophy we care about and want to win,” said Chiellini in his Press conference.“This appointment arrives mid-season, so it’s different to playing in August, but it remains an important competition and we’ve got to prove ourselves with actions rather than words, because we let a few too many of these trophies slip through our fingers in recent years and that is a shame.“We want to change that trend that has seen us lose the last two Supercoppa finals, and this is the ideal opportunity. It’s going to be a difficult match, but the objective is to start 2019 lifting a trophy above our heads.”
Police say multiple vehicles that were parked near the building were also engulfed in flames.It is believed the building was vacant and no one was inside the building or vehicles at the time of the fire.The highway was shut down temporarily and an alternate route was set up until the fire was under control.The area was secured by police pending further investigation.If you have any information, you are being asked to contact the Dawson Creek RCMP at 250-784-3700 or CrimeStoppers at 1-800-222-TIPS. POUCE COUPE, B.C. – Dawson Creek RCMP received a report of a structure fire in Pouce Coupe on the morning of June 5 at 4:10 a.m.According to RCMP, officers arrived on scene and determined that the structure on fire was located in a work yard at 5012 Highway 2.Fire crews were on scene trying to keep the fire under control.